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Geralda A. F. van Tilborg

Researcher at Eindhoven University of Technology

Publications -  16
Citations -  1339

Geralda A. F. van Tilborg is an academic researcher from Eindhoven University of Technology. The author has contributed to research in topics: Medicine & Annexin A5. The author has an hindex of 9, co-authored 10 publications receiving 1273 citations. Previous affiliations of Geralda A. F. van Tilborg include Icahn School of Medicine at Mount Sinai.

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Lipid-based nanoparticles for contrast-enhanced MRI and molecular imaging.

TL;DR: An overview of different lipidic nanoparticles for use in MRI is given, with the main emphasis on Gd–based contrast agents.
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MRI contrast agents: current status and future perspectives.

TL;DR: The current status, as well as recent and future developments of MRI contrast agents with focus on applications in oncology are described and how the basis of MR image contrast is altered by contrast agents will be discussed.
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Annexin A5-conjugated quantum dots with a paramagnetic lipidic coating for the multimodal detection of apoptotic cells

TL;DR: The specificity of the annexin A5-conjugated nanoparticles for apoptotic cells was demonstrated both with fluorescence microscopy and MRI, which confirms its potential for the detection of apoptosis with both imaging modalities in vivo.
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Annexin A5-functionalized bimodal lipid-based contrast agents for the detection of apoptosis

TL;DR: Two types of lipid-based bimodal contrast agents that enable the detection of apoptotic cells with both MRI and optical techniques are synthesized that differ both in size and in magnetic properties, which may have applications for the in vivo Detection of apoptosis.
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Annexin A5-Functionalized Bimodal Nanoparticles for MRI and Fluorescence Imaging of Atherosclerotic Plaques

TL;DR: The annexin A5-conjugated bimodal micelles displayed potential for noninvasive assessment of cell types that are considered to significantly contribute to plaque instability and therefore may be of great value in the assessment of atherosclerotic lesion phenotype.