G
Ghi Su Kim
Researcher at Asan Medical Center
Publications - 37
Citations - 877
Ghi Su Kim is an academic researcher from Asan Medical Center. The author has contributed to research in topics: Osteoporosis & Stromal cell. The author has an hindex of 16, co-authored 37 publications receiving 829 citations. Previous affiliations of Ghi Su Kim include Washington University in St. Louis & Shriners Hospitals for Children.
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Journal ArticleDOI
Antioxidant alpha-lipoic acid inhibits osteoclast differentiation by reducing nuclear factor-kappaB DNA binding and prevents in vivo bone resorption induced by receptor activator of nuclear factor-kappaB ligand and tumor necrosis factor-alpha.
TL;DR: Alpha-LA greatly suppressed in vivo bone loss induced by RANKL or TNF-alpha in a calvarial remodeling model, providing evidence that ROS plays an important role in osteoclast differentiation through NF-kappaB regulation and the antioxidant alpha-lipoic acid has a therapeutic potential for bone erosive diseases.
Journal ArticleDOI
Effects of vitamin B12 on cell proliferation and cellular alkaline phosphatase activity in human bone marrow stromal osteoprogenitor cells and UMR106 osteoblastic cells.
Ghi Su Kim,Chung Hoon Kim,Chung Hoon Kim,Jong Yeon Park,Jong Yeon Park,Kang-Sik Lee,Kang-Sik Lee,Chan-Sik Park,Chan-Sik Park +8 more
TL;DR: It is suggested that a suppressed activity of osteoblasts may contribute to osteoporosis and fractures in patients with vitamin B12 deficiency.
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Low normal TSH levels are associated with low bone mineral density in healthy postmenopausal women.
TL;DR: Whether serum TSH levels correlate with bone mineral density (BMD) is determined to determine whether hyperthyroidism and reduced bone mass are accompanied by low bone mass.
Journal ArticleDOI
Effects of dexamethasone on proliferation, activity, and cytokine secretion of normal human bone marrow stromal cells: possible mechanisms of glucocorticoid-induced bone loss.
CH Kim,SL Cheng,Ghi Su Kim +2 more
TL;DR: The results suggest that the glucocorticoid-induced bone loss is derived at least in part via inhibition of bone formation, which includes the suppression of osteoblast proliferation and collagen synthesis.
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Prevalence and associated features of albuminuria in Koreans with NIDDM.
TL;DR: The prevalence of overt proteinuria in Korean NIDDM patients with a long diabetes duration was higher than that reported in Caucasians and the data suggest that the clinical meaning of microalbuminuria may be different based on the presence or the absence of retinopathy.