G
Giampiero Pietrocola
Researcher at University of Pavia
Publications - 86
Citations - 2559
Giampiero Pietrocola is an academic researcher from University of Pavia. The author has contributed to research in topics: Bacterial adhesin & Streptococcus agalactiae. The author has an hindex of 24, co-authored 77 publications receiving 1959 citations. Previous affiliations of Giampiero Pietrocola include Polytechnic University of Milan.
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Journal ArticleDOI
Scenery of Staphylococcus implant infections in orthopedics
Lucio Montanaro,Pietro Speziale,Davide Campoccia,Stefano Ravaioli,Ilaria Cangini,Giampiero Pietrocola,Sandro Giannini,Carla Renata Arciola +7 more
TL;DR: This work reports on epidemiology of implant-related Staphylococcus infections in orthopedics, and focuses on the crucial role of bacterial adhesins and on their ability to direct the pathogenesis process.
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Protein-based biofilm matrices in Staphylococci.
TL;DR: The potential to develop vaccines to prevent protein-dependent biofilm formation during staphylococcal infection is discussed and a multitude of proteins intervene at different stages ofBiofilm formation with certain proteins contributing to biofilm accumulation and others mediating primary attachment to surfaces.
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Structural and functional role of Staphylococcus aureus surface components recognizing adhesive matrix molecules of the host
Pietro Speziale,Giampiero Pietrocola,Simonetta Rindi,Maria Provenzano,Giulio Provenza,Antonella Di Poto,Livia Visai,Carla Renata Arciola +7 more
TL;DR: These MSCRAMMs are described in terms of structural organization and ligand-binding capacity and their role as a possible target for immunotherapy is discussed.
Journal ArticleDOI
The fibrinogen receptor FbsA promotes adherence of Streptococcus agalactiae to human epithelial cells
Axel Schubert,Katherina Zakikhany,Giampiero Pietrocola,Andreas Meinke,Pietro Speziale,Bernhard J. Eikmanns,Dieter J. Reinscheid +6 more
TL;DR: It is demonstrated that FBSA promotes the adherence of S. agalactiae to epithelial cells but that FbsA does not mediate the bacterial invasion into host cells.
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Staphylococcus aureus Manipulates Innate Immunity through Own and Host-Expressed Proteases.
TL;DR: The current understanding of proteases and modulators of host proteases in the functioning of innate immunity are reviewed and the importance of these mechanisms in the pathology of staphylococcal diseases is described.