G
Giuseppe Dattilo
Researcher at University of Messina
Publications - 131
Citations - 1340
Giuseppe Dattilo is an academic researcher from University of Messina. The author has contributed to research in topics: Internal medicine & Medicine. The author has an hindex of 22, co-authored 97 publications receiving 1050 citations. Previous affiliations of Giuseppe Dattilo include Academy for Urban School Leadership.
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Journal ArticleDOI
Sodium–glucose co‐transporter 2 inhibitors in heart failure: beyond glycaemic control. A position paper of the Heart Failure Association of the European Society of Cardiology
Petar M. Seferovic,Petar M. Seferovic,Gabriele Fragasso,Mark C. Petrie,Wilfried Mullens,Roberto Ferrari,Thomas Thum,Johann Bauersachs,Stefan D. Anker,Robin Ray,Yuksel Cavusoglu,Marija Polovina,Marco Metra,Giuseppe Ambrosio,Krishna Prasad,Jelena P. Seferovic,Pardeep S. Jhund,Giuseppe Dattilo,Jelena Celutkiene,Massimo F Piepoli,Brenda Moura,Ovidiu Chioncel,Tuvia Ben Gal,Stefan Heymans,Rudolf A. de Boer,Tiny Jaarsma,Loreena Hill,Yuri Lopatin,Alexander R. Lyon,Piotr Ponikowski,Mitja Lainscak,Ewa A. Jankowska,Christian Mueller,Francesco Cosentino,Lars Lund,Gerasimos Filippatos,Frank Ruschitzka,Andrew J.S. Coats,Giuseppe M.C. Rosano +38 more
TL;DR: The present position paper summarises recent clinical trial evidence and discusses the role of SGLT2 inhibitors in the treatment of HF, pending the results of ongoing trials in different populations of patients with HF.
Journal ArticleDOI
The mosaic of the cardiac amyloidosis diagnosis: role of imaging in subtypes and stages of the disease
Gianluca Di Bella,Fausto Pizzino,Fabio Minutoli,Concetta Zito,Rocco Donato,Giuseppe Dattilo,Giuseppe Oreto,Sergio Baldari,Giuseppe Vita,Bijoy K. Khandheria,Scipione Carerj +10 more
TL;DR: The diagnostic process begins with an accurate evaluation of clinical elements and includes cardiovascular imaging, electrocardiography, serological assays, and myocardial biopsy; only the appropriate integration of these instruments can reveal the diagnosis to an expert physician.
Journal ArticleDOI
Heart Failure Association of the European Society of Cardiology update on sodium–glucose co‐transporter 2 inhibitors in heart failure
Petar M. Seferovic,Petar M. Seferovic,Gabriele Fragasso,Mark C. Petrie,Wilfried Mullens,Roberto Ferrari,Thomas Thum,Johann Bauersachs,Stefan D. Anker,Robin Ray,Yuksel Cavusoglu,Marija Polovina,Marco Metra,Giuseppe Ambrosio,Krishna Prasad,Jelena P. Seferovic,Pardeep S. Jhund,Giuseppe Dattilo,Jelena Celutkiene,Massimo F Piepoli,Brenda Moura,Ovidiu Chioncel,Tuvia Ben Gal,Stephane Heymans,Tiny Jaarsma,Loreena Hill,Yuri Lopatin,Alexander R. Lyon,Piotr Ponikowski,Mitja Lainscak,Ewa A. Jankowska,Christian Mueller,Francesco Cosentino,Lars Lund,Gerasimos Filippatos,Frank Ruschitzka,Andrew J.S. Coats,Giuseppe M.C. Rosano +37 more
TL;DR: The present document provides an update of the position paper on the role of sodium–glucose co‐transporter 2 (SGLT2) inhibitors in heart failure, based of new clinical trial evidence.
Journal ArticleDOI
Myocardial deformation in acute myocarditis with normal left ventricular wall motion--a cardiac magnetic resonance and 2-dimensional strain echocardiographic study.
Gianluca Di Bella,Michele Gaeta,Alessandro Pingitore,Giuseppe Oreto,Concetta Zito,Fabio Minutoli,Carmelo Anfuso,Giuseppe Dattilo,Annalisa Lamari,Sebastiano Coglitore,Scipione Carerj +10 more
TL;DR: In this paper, a delayed enhancement (DE) CMR study was performed to identify damaged myocardial segments in patients with acute myocarditis and showed a significant reduction in the longitudinal deformation of the left ventricle.
Journal ArticleDOI
Left Atrial Morphology, Size and Function in Patients With Transthyretin Cardiac Amyloidosis and Primary Hypertrophic Cardiomyopathy - Comparative Strain Imaging Study.
Cesare de Gregorio,Giuseppe Dattilo,Matteo Casale,Anna Terrizzi,Rocco Donato,Gianluca Di Bella +5 more
TL;DR: LA reservoir and pump function were significantly impaired in both TTR-CA and HCM patients compared with controls, but mainly in the former group, irrespective of LA volume and LV ejection fraction, likely caused by a more altered LA wall structure.