G
Gokhan S. Kilic
Researcher at University of Texas Medical Branch
Publications - 87
Citations - 1538
Gokhan S. Kilic is an academic researcher from University of Texas Medical Branch. The author has contributed to research in topics: Hysterectomy & Retrospective cohort study. The author has an hindex of 22, co-authored 81 publications receiving 1280 citations. Previous affiliations of Gokhan S. Kilic include University of Texas at Austin & University of Texas Southwestern Medical Center.
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Journal ArticleDOI
Signaling Pathways in Leiomyoma: Understanding Pathobiology and Implications for Therapy
TL;DR: These pathways are dissected and their interconnections, aberrations and role in leiomyoma pathobiology are discussed, to identify potential targets for development of novel therapeutics and underlines the multifactorial origin and complex nature of these tumors.
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Estrogen Receptors and Signaling in Fibroids: Role in Pathobiology and Therapeutic Implications.
TL;DR: The types and structure of estrogen receptors (nuclear and membrane bound, including α and β receptors and G protein-coupled estrogen receptor 1 GPER1) and estrogen-signaling pathways in fibroids are discussed.
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From endometrial hyperplasia to endometrial cancer: insight into the biology and possible medical preventive measures
TL;DR: Biomarkers whose expression is altered in cases of endometrial hyperplasia or cancer such as progesterone receptor, insulin-like growth factor I, retinaldehyde dehydrogenase type II, and secreted frizzled-related protein 4, seem to be promising to use as early-stage tumor markers.
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Trends in the national distribution of laparoscopic hysterectomies from 2003 to 2010.
Jinhyung Lee,Kristofer Jennings,Mostafa A. Borahay,Ana M. Rodriguez,Gokhan S. Kilic,Russell R. Snyder,Pooja R. Patel +6 more
TL;DR: There remains a gap in distribution of laparoscopic hysterectomies with regards to age, race, median income and insurance type that does not seem to be closing, despite the increased availability of Laparoscopic HystereCTomies.
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Expression of dioxin-related transactivating factors and target genes in human eutopic endometrial and endometriotic tissues
TL;DR: The proposed link between dioxin exposure and endometriosis may be explained in part by the up-regulation of the CYP1A1 gene expression in endometiotic tissues, which may give rise to significantly increased P-4501A1 enzyme activity and promote the development and growth of endometRIosis.