Gonzalo A. Carrasco

TL;DR: The studies described in this review suggest that multiple brain mechanisms are responsible for the regulation of each hormone and that not all hormones are regulated by the same neural circuits.

TL;DR: In this paper, the authors examined behavioral responses to a pleasurable stimulus (sucrose), estrous cycle length (in female rats), and plasma hormone levels following systemic administration of a selective 5-HT1A receptor agonist [(+)8-hydroxy-N,N-dipropyl-2-aminotetralin hydrobromide (8-OH-DPAT); 40μg/kg, s.c., administered 15min prior to sacrifice], in male and female rats exposed to 4 weeks of CMS.

TL;DR: On page 4 of this published article an error in Figure 2(a) has occurred and it has been replaced with a corrected version of the figure as shown below.

TL;DR: Data provide the first direct evidence that neuroendocrine responses to a peripheral injection of (−)DOI are predominantly mediated by activation of 5-HT2A receptors in the hypothalamic paraventricular nucleus.

TL;DR: This study indicates that a putative extra-nuclear estrogen receptor, GPR30, may play a role in estradiol-mediated attenuation of 5-HT(1A) receptor signaling, and potentially in accelerating the effects of SSRIs in treatment of mood disorders.