Louis D. Van de Kar

TL;DR: The studies described in this review suggest that multiple brain mechanisms are responsible for the regulation of each hormone and that not all hormones are regulated by the same neural circuits.

TL;DR: The combined neuroanatomical and pharmacological observations suggest that the neuroendocrine responses to DOI are mediated by activation of neurons in the hypothalamic paraventricular nucleus and associated circuitry.

TL;DR: The results indicate that lack of the 5-HTT is associated with a functional desensitization of 5- HT(1A) receptor responses to 8-OH-DPAT, which may be a consequence, at least in part, of the decrease in density of5-HT( 1A) receptors in the hypothalamus and dorsal raphe of 5 -HTT-/- mice.

TL;DR: In this paper, the authors examined behavioral responses to a pleasurable stimulus (sucrose), estrous cycle length (in female rats), and plasma hormone levels following systemic administration of a selective 5-HT1A receptor agonist [(+)8-hydroxy-N,N-dipropyl-2-aminotetralin hydrobromide (8-OH-DPAT); 40μg/kg, s.c., administered 15min prior to sacrifice], in male and female rats exposed to 4 weeks of CMS.

TL;DR: It is suggested that chronic, but not acute, exposure to fluoxetine decreases 5-HT1A receptor function, which is not seen in rats chronically exposed to DMI.