G
Gordon H. Williams
Researcher at Brigham and Women's Hospital
Publications - 563
Citations - 39948
Gordon H. Williams is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Angiotensin II & Aldosterone. The author has an hindex of 88, co-authored 559 publications receiving 38048 citations. Previous affiliations of Gordon H. Williams include University of Sydney & University of Michigan.
Papers
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Acute and delayed effects of corticotropin-releasing hormone on dopamine activity in man
Joel A. Posener,Joseph J. Schildkraut,Gordon H. Williams,Ray E. Gleason,Melinda S. Salomon,Gabriel Mecheri,Alan F. Schatzberg +6 more
TL;DR: The findings of these pilot studies suggest that CRH may exert delayed but not acute effects on dopamine activity in man.
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Decreased Adrenal Responsiveness to Angiotensin II: A Defect Present in Spontaneously Hypertensive Rats: A POSSIBLE MODEL OF HUMAN ESSENTIAL HYPERTENSION
TL;DR: The SHR PRA and aldosterone responses to sodium restriction and a Aldosterone response to A II were similar to that previously described in a subgroup of patients with essential hypertension suggesting that the SHR will serve as a model for exploring the mechanism(s) responsible for the hypertension in these patients.
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Glycated albumin and direct low density lipoprotein cholesterol levels in type 2 diabetes mellitus.
Masumi Ai,Seiko Otokozawa,Ernst J. Schaefer,Bela F. Asztalos,Katsuyuki Nakajima,Peter Shrader,Sekar Kathiresan,James B. Meigs,Gordon H. Williams,Gordon H. Williams,David M. Nathan +10 more
TL;DR: Calculated HDL-C provides an accurate assessment of fasting LDL-C compared with a direct measurement in most subjects, except for those with hypertriglyceridemia, and GA correlates with HbA1c in diabetic and non-diabetic subjects and may serve as a reasonable marker of short term diabetic control.
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Essential hypertension as an endocrine disease.
TL;DR: Several lines of evidence strongly suggest that the majority of patients with essential hypertension have an endocrine basis for their elevated blood pressure, and there is a high likelihood that a genetic marker can be found to identify each subgroup.
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Genetic approach to diagnostic and therapeutic decisions in human hypertension.
TL;DR: The most convincing evidence supports a role for the angiotensinogen gene, where linkage has been documented and an association with an intermediate phenotype of hypertension (nonmodulation) has been reported.