H
Hadi Fathi Moghaddam
Researcher at Ahvaz Jundishapur University of Medical Sciences
Publications - 21
Citations - 840
Hadi Fathi Moghaddam is an academic researcher from Ahvaz Jundishapur University of Medical Sciences. The author has contributed to research in topics: Oxidative stress & Venlafaxine. The author has an hindex of 9, co-authored 19 publications receiving 585 citations.
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Pathogenic mechanisms following ischemic stroke
TL;DR: The underlying pathophysiology of ischemic stroke is reviewed and the intertwined pathways that are promising therapeutic targets are revealed, leading to the development of numerous agents that target various injury pathways.
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Emerging Roles of microRNAs in Ischemic Stroke: As Possible Therapeutic Agents.
TL;DR: The role of miRNAs are focused on as potential diagnostic and prognostic biomarkers, as well as promising therapeutic agents in cerebral ischemic stroke.
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Protective effects of gallic acid against chronic cerebral hypoperfusion-induced cognitive deficit and brain oxidative damage in rats
Mehrdad Shahrani Korani,Yaghoub Farbood,Alireza Sarkaki,Hadi Fathi Moghaddam,Mohammad Taghi Mansouri +4 more
TL;DR: Gallic acid has beneficial activity against 2VO-induced cognitive deficits via enhancement of cerebral antioxidant defense and the present study suggested that GA might be useful in the treatment of VD.
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Anti-diabetic effect of betulinic acid on streptozotocin-nicotinamide induced diabetic male mouse model
TL;DR: In this article, the effects of betulinic acid (BA, a triterpene) are evaluated on glucose, α-amylase and plasma insulin levels, insulin resistance and the histopathology of pancreatic islets in diabetic mice.
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The effect of hydro-alcoholic celery (Apiumgraveolens) leaf extract on cardiovascular parameters and lipid profile in animal model of hypertension induced by fructose.
TL;DR: Celery leaf extract with its blood pressure and lipid lowering effects, can be considered as an antihypertensive agent in chronic treatment of elevated SBP in animals’ model of hypertension induced by fructose.