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Hamid Najafipour

Researcher at Kerman Medical University

Publications -  160
Citations -  1951

Hamid Najafipour is an academic researcher from Kerman Medical University. The author has contributed to research in topics: Medicine & Population. The author has an hindex of 21, co-authored 128 publications receiving 1507 citations. Previous affiliations of Hamid Najafipour include University of Glasgow.

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Circulating levels of interleukin-35 in patients with multiple sclerosis: evaluation of the influences of FOXP3 gene polymorphism and treatment program.

TL;DR: Higher serum levels of IL-35 in treated MS patients showed that the benefit effects of treatment may in part performed through the upregulation of the IL- 35 production, suggesting that the SNP rs3761548 may influence the susceptibility to MS disease and the serum levels.
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The Effect of Saffron Consumption on Biochemical and Histopathological Heart Indices of Rats with Myocardial Infarction

TL;DR: Saffron + ISO group showed remarkably decreased intensity of tissue destruction and significantly decreased serum levels of heart troponin I, when compared to ISO group, suggesting the protective role of saffron on ischemic hearts by biochemical and histopathological findings.
Journal Article

Coronary Artery Disease Risk Factors in an Urban and Peri-urban Setting, Kerman, Southeastern Iran (KERCADR Study): Methodology and Preliminary Report.

TL;DR: The first round of the KERCADR cohort with sufficient sample size and response rate provided precise estimates for the main clinical and para-clinical NCD risk factors, which need to be translated into public health interventions and monitored in the next rounds of the cohort.
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Changes in synovial PO2 and blood flow in the rabbit knee joint due to stimulation of the posterior articular nerve

TL;DR: In view of the low Ps,O2 values occurring deep within the joint, avascular structures such as cartilage could be subject to injury if sustained reduction in synovial blood flow occurred, which could be a contributory factor in the pathogenesis of degenerative and inflammatory joint diseases.
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Effect of melatonin on intracranial pressure and brain edema following traumatic brain injury: role of oxidative stresses.

TL;DR: Melatonin decreases brain edema, BBB permeability and ICP, but increases VCS after TBI, probably due to inhibition of oxidative stress.