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Harold N. Trick

Researcher at Kansas State University

Publications -  96
Citations -  7983

Harold N. Trick is an academic researcher from Kansas State University. The author has contributed to research in topics: Gene & Transformation (genetics). The author has an hindex of 40, co-authored 88 publications receiving 6777 citations. Previous affiliations of Harold N. Trick include Ohio Agricultural Research and Development Center & Ohio State University.

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A gene in the multidrug and toxic compound extrusion (MATE) family confers aluminum tolerance in sorghum.

TL;DR: Positional cloning is used to identify the gene encoding a member of the multidrug and toxic compound extrusion (MATE) family, an aluminum-activated citrate transporter, as responsible for the major sorghum aluminum tolerance locus, AltSB, and find superior AltSB haplotypes that can be incorporated via molecular breeding and biotechnology into acid soil breeding programs, thus helping to increase crop yields in developing countries where acidic soils predominate.
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Molecular Characterization of the Major Wheat Domestication Gene Q

TL;DR: Rachis fragility, glume shape, and glume tenacity mimicked the q phenotype in transgenic plants exhibiting post-transcriptional silencing of the transgene and the endogenous Q gene, andVariation in spike compactness and plant height were associated with the level of transGene transcription due to the dosage effects of Q.
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Map-based cloning of leaf rust resistance gene Lr21 from the large and polyploid genome of bread wheat.

TL;DR: The map-based cloning of the leaf rust resistance gene Lr21, previously mapped to a gene-rich region at the distal end of chromosome arm 1DS of bread wheat, opens the door for cloning of many crop-specific agronomic traits located in the gene- rich regions of bread Wheat.
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Genetically engineered resistance to Fusarium head blight in wheat by expression of Arabidopsis NPR1.

TL;DR: It is suggested that FHB resistance in the AtNPR1-expressing wheat is a result of these plants being more responsive to an endogenous activator of plant defense, and it is demonstrated that NPR1 is an effective candidate for controlling FHB.