Hartwell H. Whisennand

TL;DR: A device consisting of a highly differentiated human liver cell line cultured in a hollow fiber cartridge is capable of supporting dogs with acetaminopheninduced fulminant hepatic failure for a period long enough for their own livers to resume function, allowing recovery of hepatocytes that would otherwise have lysed.

TL;DR: The results of this study suggest the need for avoidance of technical problems during operations, earlier referral of patients with known abdominal aortic aneurysms, especially the elderly, and early diagnosis with immediate operation for ruptured aneurYSms.

TL;DR: An improved model of fulminant hepatic failure in dogs is established, using Buthionine sulfoximine to inactivate glutathione synthesis, and small increments of acetaminophen given intravenously to maintain the plasma level at approximately 200 μg/ml for 20 hr.

TL;DR: Among 76 patients with heart wounds treated over a 4 year period, nine had penetrating injuries to the coronary arteries with clinical presentations of pericardial tamponade, electrocardiographic abnormalities of bundle branch block or ST and T wave changes, and hemothorax.

TL;DR: This case describes a patient with cholesteryl ester storage disease who underwent liver transplantation for progressive cirrhosis, portal hypertension, ascites, and uncontrollable gastrointestinal bleeding who is clinically well except for mild hypersplenism and an elevated blood urea nitrogen (BUN) and creatinine.