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Haw Ming Huang

Researcher at Taipei Medical University

Publications -  99
Citations -  1532

Haw Ming Huang is an academic researcher from Taipei Medical University. The author has contributed to research in topics: Chemistry & Medicine. The author has an hindex of 18, co-authored 84 publications receiving 1182 citations.

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Resonance frequency assessment of dental implant stability with various bone qualities: a numerical approach.

TL;DR: The results suggest that RFA could serve as a non-invasive diagnostic tool for detecting the stability of dental implants during the healing stages and in subsequent routine follow-up care after treatment.
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Early detection of implant healing process using resonance frequency analysis.

TL;DR: In vitro and in vivo models were adopted for assessing the application of resonance frequency analysis (RFA) in the early detection of implant stability and it was concluded that RFA is a reliable and accurate method for early assessment of the osseointegration process.
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Static Magnetic Fields Promote Osteoblast-Like Cells Differentiation Via Increasing the Membrane Rigidity

TL;DR: One of the possible mechanisms that SMF affects osteoblastic maturation is by increasing the membrane rigidity and reducing the proliferation-promoting effects of growth factors at the membrane domain, according to the authors.
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Static magnetic fields up-regulate osteoblast maturity by affecting local differentiation factors.

TL;DR: Evidence is provided that static magnetic fields affect osteoblastic maturation by up-regulating early local factors that produce morphologic changes and matrix vesicles release in MG63 osteoblast-like cells.
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Static magnetic fields enhance dental pulp stem cell proliferation by activating the p38 mitogen-activated protein kinase pathway as its putative mechanism

TL;DR: Investigating the effect of SMFs on DPSC proliferation and its possible intracellular mechanism of action suggested that the p38 signalling cascade was activated when the DPSCs were exposed to a 0.4‐T SMF and may activate p38 mitogen‐activated protein kinase signalling, and thus reorganize the cytoskeleton, which contributes to the increased cell proliferation of the DPSC.