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Heini Saarimäki

Researcher at Aalto University

Publications -  19
Citations -  960

Heini Saarimäki is an academic researcher from Aalto University. The author has contributed to research in topics: Functional magnetic resonance imaging & Brain activity and meditation. The author has an hindex of 10, co-authored 18 publications receiving 731 citations. Previous affiliations of Heini Saarimäki include University of Edinburgh.

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Discrete Neural Signatures of Basic Emotions

TL;DR: Brain regions contributing most to the classification accuracy included medial and inferior lateral prefrontal cortices, frontal pole, precentral and postcentral gyri, precuneus, and posterior cingulate cortex, suggesting a direct link between activity in these brain regions and the subjective emotional experience.
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Emotional speech synchronizes brains across listeners and engages large-scale dynamic brain networks

TL;DR: It is proposed that high arousal synchronizes the listeners' sound-processing and speech-comprehension networks, whereas negative valence synchronizes circuitries supporting emotional and self-referential processing.
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Distributed affective space represents multiple emotion categories across the human brain.

TL;DR: It is concluded that different basic and non-basic emotions have distinguishable neural bases characterized by specific, distributed activation patterns in widespread cortical and subcortical circuits.
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Effects of spatial smoothing on functional brain networks

TL;DR: In this article, the authors apply various levels of spatial smoothing to resting-state fMRI data and measure the changes induced in functional networks, showing that the level of smoothing clearly affects the degrees and other centrality measures of functional network nodes; these changes are non-uniform, systematic, and depend on the geometry of the brain.
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Memory binding and white matter integrity in familial Alzheimer’s disease

TL;DR: The results indicate that white matter structures in the frontal and temporal lobes are vulnerable to the early stages of familial Alzheimer's disease and their damage is associated with impairments in two memory binding functions known to be markers for Alzheimers disease.