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Henry S. Loeb

Researcher at United States Department of Veterans Affairs

Publications -  91
Citations -  10193

Henry S. Loeb is an academic researcher from United States Department of Veterans Affairs. The author has contributed to research in topics: Myocardial infarction & Heart failure. The author has an hindex of 31, co-authored 91 publications receiving 10033 citations. Previous affiliations of Henry S. Loeb include University of Minnesota & Argonne National Laboratory.

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A Comparison of Enalapril with Hydralazine–Isosorbide Dinitrate in the Treatment of Chronic Congestive Heart Failure

TL;DR: The different effects of the two regimens (enalapril and hydralazine-isosorbide dinitrate) on mortality and physiologic end points suggest that the profile of effects might be enhanced if the regimens were used in combination.
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Effect of vasodilator therapy on mortality in chronic congestive heart failure. Results of a Veterans Administration Cooperative Study.

TL;DR: The data suggest that the addition of hydralazine and isosorbide dinitrate to the therapeutic regimen of digoxin and diuretics in patients with chronic congestive heart failure can have a favorable effect on left ventricular function and mortality.
Journal Article

Ejection fraction, peak exercise oxygen consumption, cardiothoracic ratio, ventricular arrhythmias, and plasma norepinephrine as determinants of prognosis in heart failure

TL;DR: In this article, the influence of prerandomization measurements on subsequent mortality was examined using data from the Department of Veterans Affairs Cooperative Vasodilator-Heart Failure Trials (V-HeFT I, 641 patients; V-HEFT II, 804 patients).
Journal Article

Ejection fraction, peak exercise oxygen consumption, cardiothoracic ratio, ventricular arrhythmias, and plasma norepinephrine as determinants of prognosis in heart failure. The V-HeFT VA Cooperative Studies Group

TL;DR: Optimal assessment of the mortality risk in an individual or a group of individuals with heart failure uses measurement of EF, peak Vo2, CTR, plasma norepinephrine, and the presence of ventricular arrhythmias.