H
Hermann Steller
Researcher at Rockefeller University
Publications - 121
Citations - 21474
Hermann Steller is an academic researcher from Rockefeller University. The author has contributed to research in topics: Programmed cell death & Apoptosis. The author has an hindex of 64, co-authored 120 publications receiving 20320 citations. Previous affiliations of Hermann Steller include University of California, Berkeley & Massachusetts Institute of Technology.
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Positional information along the dorsal-ventral axis of the drosophila eye: graded expression of the four-jointed gene
Michael Brodsky,Hermann Steller +1 more
TL;DR: Analysis of the fj phenotype and expression pattern in the leg suggests that fj is required for cell-cell signaling during disc development, and a systematic screen for P-element insertions that show nonuniform reporter gene expression along this axis found patterns of transcriptional enhancer activity established early in disc development.
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Unfolded protein response in Gaucher disease: From human to Drosophila
TL;DR: The results strongly suggest that mutant GCase induces the UPR in GD patients as well as in carriers of GD mutations and leads to development of locomotion deficit in flies heterozygous for GD mutations.
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Facing death in the fly: genetic analysis of apoptosis in Drosophila
Kimberly McCall,Hermann Steller +1 more
TL;DR: Through the combined use of genetic, molecular, biochemical and cell biological techniques in Drosophila it should now be possible to elucidate the precise mechanism by which apoptosis occurs, and how the death program is activated in response to many distinct death-inducing signals.
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Migration of glial cells into retinal axon target field in Drosophila melanogaster.
Sharon E. Perez,Hermann Steller +1 more
TL;DR: This study defines the origin of lamina glia (L glia), and demonstrates that L glia migrate into the lamina over a considerable distance, consistent with a role for the glia in providing guidance cues to the R axons.
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The Drosophila caspases Strica and Dronc function redundantly in programmed cell death during oogenesis.
TL;DR: A novel pathway of cell death in the ovary is revealed that utilizes strica, dronc, dcp-1 and drice, and importantly illustrates strong redundancy among the caspases, which indicates that caspase are required for nurse cell death during mid-oogenesis, and participate in developmental nurse cellDeath during late oogenesis.