H
Hermann Steller
Researcher at Rockefeller University
Publications - 121
Citations - 21474
Hermann Steller is an academic researcher from Rockefeller University. The author has contributed to research in topics: Programmed cell death & Apoptosis. The author has an hindex of 64, co-authored 120 publications receiving 20320 citations. Previous affiliations of Hermann Steller include University of California, Berkeley & Massachusetts Institute of Technology.
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Journal ArticleDOI
The Sept4 Septin Locus Is Required for Sperm Terminal Differentiation in Mice
Holger Kissel,Maria-Magdalena Georgescu,Sarit Larisch,Katia Manova,Gary R. Hunnicutt,Hermann Steller +5 more
TL;DR: It is shown that Sept4 proteins play distinct but evolutionarily conserved functions in different cellular compartments, consistent with a role of the proapoptotic Sept4 protein ARTS in promoting caspase-mediated removal of cytoplasm via inhibition of XIAP.
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A novel mitochondrial septin-like protein, ARTS, mediates apoptosis dependent on its P-loop motif.
Sarit Larisch,Youngsuk Yi,Rona Lotan,Hedviga Kerner,Sarah Eimerl,W. Tony Parks,Yossi Gottfried,Stephanie Birkey Reffey,Mark P. de Caestecker,David Danielpour,Naomi Book-Melamed,Rina Timberg,Colin S. Duckett,Robert J. Lechleider,Robert J. Lechleider,Hermann Steller,Joseph Orly,Seong-Jin Kim,Anita B. Roberts +18 more
TL;DR: A protein product of the human septin H5/PNUTL2/CDCrel2b gene called ARTS (for apoptosis-related protein in the TGF-β signalling pathway) was described in this article.
Journal ArticleDOI
A transposable P vector that confers selectable G418 resistance to Drosophila larvae.
Hermann Steller,V. Pirrotta +1 more
TL;DR: A vector for P‐mediated transformation is constructed which uses the finding that the neo gene transcribed from the herpes thymidine kinase promoter gives low levels of resistance but high levels can be obtained using the hsp70 heat‐shock promoter.
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Regulation of apoptosis in Drosophila
TL;DR: Reaper-family proteins are produced, which promote the auto-ubiquitination and degradation of Diap1, thereby removing the ‘brakes on death’ in cells that are doomed to die, indicating that the control of cell survival and death relies extensively on targeted degradation by the ubiquitin–proteasome pathway.
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disconnected: A locus required for neuronal pathway formation in the visual system of drosophila
TL;DR: The different aspects of the phenotype suggest that the disconnected gene plays a role in neuronal cell recognition, and in a subset of other peripheral neurons in the thoracic and abdominal segments.