H
Huan Jun Lu
Researcher at Yanbian University
Publications - 7
Citations - 20
Huan Jun Lu is an academic researcher from Yanbian University. The author has contributed to research in topics: Medicine & Nociception. The author has an hindex of 1, co-authored 1 publications receiving 3 citations.
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Journal ArticleDOI
Astrocytes in Chronic Pain: Cellular and Molecular Mechanisms
Huan Jun Lu,Yongjing Gao +1 more
TL;DR: In this paper , the authors present the current understanding of the roles of astrocytes in chronic pain, how they regulate nociceptive responses, and their cellular and molecular mechanisms of action.
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Enhanced function of NR2C/2D-containing NMDA receptor in the nucleus accumbens contributes to peripheral nerve injury-induced neuropathic pain and depression in mice
TL;DR: Results for the first time showed that the functional change of NR2C/2D subunits-containing NMDARs in the NAc might contribute to the sensory and affective components in neuropathic pain.
Journal ArticleDOI
Progress on the Elucidation of the Antinociceptive Effect of Ginseng and Ginsenosides in Chronic Pain
Mei-Xian Li,Qian Wei,Huan Jun Lu +2 more
TL;DR: This review presents the current understanding of the effectiveness of ginseng in chronic pain and how its active constituents regulate nociceptive responses and their mechanisms of action.
Journal ArticleDOI
Functional Connections of the Vestibulo-spino-adrenal Axis in the Control of Blood Pressure Via the Vestibulosympathetic Reflex in Conscious Rats.
TL;DR: Results indicate that the VSA axis may be a key component of the pathway used by the vestibulosympathetic reflex to maintain blood pressure during postural movements.
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Ginsenoside Rh2 Ameliorates Neuropathic Pain by inhibition of the miRNA21-TLR8-mitogen-activated protein kinase axis
TL;DR: In this article , the effects of ginsenoside Rh2 on spared nerve injury (SNI)-induced neuropathic pain and elucidated the potential mechanisms were investigated and various doses of rh2 intrathecal injection dose-dependently attenuated SNI-induced mechanical allodynia and thermal hyperalgesia.