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Ida J. Llewellyn-Smith
Researcher at Flinders University
Publications - 134
Citations - 6028
Ida J. Llewellyn-Smith is an academic researcher from Flinders University. The author has contributed to research in topics: Spinal cord & Rostral ventrolateral medulla. The author has an hindex of 43, co-authored 134 publications receiving 5734 citations. Previous affiliations of Ida J. Llewellyn-Smith include University of Auckland & University of Oxford.
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Projections of substance P-containing neurons within the guinea-pig small intestine.
M. Costa,M. Costa,John B. Furness,John B. Furness,Ida J. Llewellyn-Smith,Ida J. Llewellyn-Smith,A.C. Cuello,A.C. Cuello +7 more
TL;DR: The origins of substance P immunoreactive axons in the small intestine of the guinea-pig were investigated with an immunohistochemical technique in whole mount preparations and it is likely that the highly organised patterns of innervation by different substance P-containing neurons have specific roles in the intestine.
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Subgroups of hindbrain catecholamine neurons are selectively activated by 2-deoxy-d-glucose induced metabolic challenge
TL;DR: The predominant and seemingly preferential activation of epinephrine neurons suggests that they may play a unique role in the brain's response to glucose deficit.
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Ultrastructural localization of nitric oxide synthase immunoreactivity in guinea-pig enteric neurons.
TL;DR: Results indicate that nitric oxide may regulate the activity of both myenteric neurons and smooth muscle, and leave unanswered the question of how Nitric oxide is stored and released.
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Neurochemically similar myenteric and submucous neurons directly traced to the mucosa of the small intestine.
TL;DR: It has been possible for the first time to trace the processes of chemically specified neurons through the layers of the intestinal wall and to show by a direct method that CGRP/CCK/ChAT/NPY/ SOM myenteric and submucous nerves cells provide terminals in the mucosa.
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Innocuous, not noxious, input activates PKCgamma interneurons of the spinal dorsal horn via myelinated afferent fibers.
TL;DR: It is established that PKCγ interneurons are activated by myelinated afferents that respond to innocuous stimuli, which suggests that injury-induced mechanical allodynia is transmitted through a circuit that involves PKCα interneURons and non-nociceptive, VGLUT1-expressing myelination primary afferentS.