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Isabelle Vaillant

Researcher at University of Auvergne

Publications -  16
Citations -  1434

Isabelle Vaillant is an academic researcher from University of Auvergne. The author has contributed to research in topics: Heterochromatin & DNA methylation. The author has an hindex of 11, co-authored 16 publications receiving 1278 citations. Previous affiliations of Isabelle Vaillant include Centre national de la recherche scientifique & University of Geneva.

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An siRNA pathway prevents transgenerational retrotransposition in plants subjected to stress

TL;DR: It is found that natural and experimentally induced variants in ONSEN insertions confer heat responsiveness to nearby genes, and therefore mobility bursts may generate novel, stress-responsive regulatory gene networks.
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Stress-Induced Activation of Heterochromatic Transcription

TL;DR: It is found that heterochromatin-associated silencing in Arabidopsis plants subjected to a particular temperature regime is released in a genome-wide manner without alteration of repressive epigenetic modifications and does not involve common epigenetic mechanisms.
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Role of histone and DNA methylation in gene regulation.

TL;DR: This work has suggested that maintenance of DNA methylation patterns at CG sequences is responsible for the formation of stable and thus heritable activity states termed epialleles.
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Changes in 5S rDNA Chromatin Organization and Transcription during Heterochromatin Establishment in Arabidopsis

TL;DR: It is shown that 5S rDNA methylation is reduced in 2- day-old seedlings versus 4-day-old or older aerial plant tissues, and the minor5S rRNA species are expressed most abundantly at this stage, and that in leaf nuclei of mature wild-type plants, the transcribed fraction of 5 S rDNA forms loops that emanate from chromocenters.
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MOM1 mediates DNA‐methylation‐independent silencing of repetitive sequences in Arabidopsis

TL;DR: It is shown that mutation of MORPHEUS’ MOLECULE 1 (MOM1) releases 5S repeat silencing independently of chromatin properties, as illustrated by the absence of detectable alteration of DNA and histone H3 methylation patterns.