Showing papers by "Istvan Jona published in 2014"

TL;DR: Eu3+ was tested on single RyR1 channels reconstituted into planar lipid bilayers and found that RyR open probability was insensitive to lowering [Ca2+] to 100 nM as long as the toxin occluded the vestibule, suggesting that Eu3 + acts as an agonist of both Ca2+-binding sites.