J.J. Bouyer

TL;DR: Comparison of the effects on sleep of a 1 h-lasting restraint stress applied at light onset to those observed after the same stressor was applications at light offset shows that restraint stress could be efficiently used to study the interaction between the circadian and homeostatic components of sleep regulation.

TL;DR: The validity and suitability of a rodent model for studying the effects of noise on sleep and definitively show that sleep is disturbed by EN exposure are demonstrated.

TL;DR: The results indicate that the response of the sleep-wakefulness cycle to stress is related to the behavioral reactivity to stress, in turn governed by the individual's reactivity of the HPA axis.

TL;DR: Results show that a chronic exposure to EN restricts continually amounts of slow wave sleep (SWS) and paradoxical sleep (PS) and fragments these two sleep stages with no habituation effect, and suggests that this psychobiological profile of subjects could be responsible for their SWS vulnerability under a chronic EN exposure.

TL;DR: This work investigated whether neurosteroids infused into the nucleus basalis magnocellularis affected the sleep-wakefulness cycle in rats, measured by electroencephalographic recordings and showed that pregnenolone sulfate increased, whereas allopregnanolone decreased, the duration of paradoxical sleep in the 24 h interval following injection compared to control recordings.