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J K Park

Researcher at Chonbuk National University

Publications -  8
Citations -  86

J K Park is an academic researcher from Chonbuk National University. The author has contributed to research in topics: Dopamine receptor & Angiotensin II receptor type 1. The author has an hindex of 6, co-authored 8 publications receiving 83 citations.

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Role of rho-kinase activity in angiotensin II-induced contraction of rabbit clitoral cavernosum smooth muscle.

TL;DR: It is demonstrated that G-protein-coupled increase in myofilament Ca2+ sensitivity mediated through the RhoA/Rho-kinase signal pathway is involved in the control by ANG II of the clitoral CSM tone.
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Implantation of penile prosthesis in a patient with severe corporeal fibrosis induced by cavernosal injection therapy.

TL;DR: A new technique using electric resection and electrovaporization of fibrotic tissue for implantation of penile prosthesis in a patient with severe corporeal fibrosis is introduced.
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The prevalence of causative organisms of community-acquired urethritis in an age group at high risk for sexually transmitted infections in Korean Soldiers

TL;DR: CT was the most prevalent STI pathogen and coinfections of NG with non-NG appeared less frequently, and the young male soldiers with urethritis should be administered suitable antibiotics for STI pathogens that were found by mPCR results, rather than an experimental combination of antibiotics for coinfection.
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The effects of dopamine receptor agonists on BK Ca channels and signal transduction mechanism in corpus cavernosal smooth muscle cells.

TL;DR: Enzyme immunoassay indicated that dopamine and apomorphine stimulates cAMP levels in corporal smooth muscle cells in a concentration-dependent fashion, suggesting that the activation of BKCa channels by dopamine receptor agonists in Corporal smooth Muscle cells might be one of the mechanisms in inducing penile erection.
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Effects of ginsenoside on large-conductance K Ca channels in human corporal smooth muscle cells

TL;DR: The results of this study suggested that the activation of large-conductance KCa channels by ginsenoside could be one mechanism of gINSenoside-induced relaxation in corporal smooth muscle.