J
James D. Dick
Researcher at Johns Hopkins University
Publications - 85
Citations - 6224
James D. Dick is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Vancomycin & Bacteremia. The author has an hindex of 40, co-authored 85 publications receiving 5983 citations. Previous affiliations of James D. Dick include University of Maryland, College Park & Johns Hopkins University School of Medicine.
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Journal ArticleDOI
Fatty acid synthesis: a potential selective target for antineoplastic therapy
Francis P. Kuhajda,Kris Jenner,Fawn D. Wood,Randolph A. Hennigar,Randolph A. Hennigar,Lisa B. Jacobs,James D. Dick,Gary R. Pasternack +7 more
TL;DR: Tumor cell lines with elevated fatty acid synthase showed commensurate increases in incorporation of [U-14C]acetate into acylglycerols demonstrating that fatty acids synthase increases occur in the context of overall increases in endogenous fatty acid synthesis.
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Mechanisms of latency in Mycobacterium tuberculosis
TL;DR: The mechanisms by which M. tuberculosis establishes a latent metabolic state, eludes immune surveillance and responds to triggers that stimulate reactivation are a high priority for the future control of TB.
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Failure of clindamycin treatment of methicillin-resistant Staphylococcus aureus expressing inducible clindamycin resistance in vitro.
TL;DR: A case of a surgical site infection caused by clindamycin-susceptible, erythromycin-resistant methicillin-resistant Staphylococcus aureus (MRSA) that did not respond to treatment with clindycin is reported.
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Vancomycin Resistance in Staphylococci
TL;DR: This article reviews the major epidemiologic, microbiologic, and clinical characteristics of vancomycin resistance in both coagulase-negative staphylococci and S. aureus and discusses issues unique to each organism.
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Septic reactions to platelet transfusions. A persistent problem.
TL;DR: Clinically, septic reactions were associated with greater temperature elevations than febrile reactions to sterile products, and contamination of platelet concentrates remains a significant clinical problem.