J
James N. Cobley
Researcher at University of the Highlands and Islands
Publications - 44
Citations - 2024
James N. Cobley is an academic researcher from University of the Highlands and Islands. The author has contributed to research in topics: Oxidative stress & Medicine. The author has an hindex of 20, co-authored 37 publications receiving 1468 citations. Previous affiliations of James N. Cobley include Abertay University & Liverpool John Moores University.
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13 reasons why the brain is susceptible to oxidative stress.
TL;DR: 13 reasons why the brain is susceptible to oxidative stress are rationalised and key reasons include inter alia unsaturated lipid enrichment, mitochondria, calcium, glutamate, modest antioxidant defence, redox active transition metals and neurotransmitter auto-oxidation.
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Assessment of vitamin D concentration in non-supplemented professional athletes and healthy adults during the winter months in the UK: implications for skeletal muscle function
Graeme L. Close,J. Russell,James N. Cobley,Daniel J. Owens,George S. Wilson,Warren Gregson,William D. Fraser,James P. Morton +7 more
TL;DR: The current data supports previous findings that athletes living at Northerly latitudes (UK = 53° N) exhibit inadequate vitamin D concentrations and suggests that inadequateitamin D concentration is detrimental to musculoskeletal performance in athletes.
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Exercise-induced muscle damage: What is it, what causes it and what are the nutritional solutions?
TL;DR: A pragmatic practical summary that can be adopted by practitioners and direct future research is presented, with the purpose of pushing the field to better consider the fine balance between recovery and adaptation and the potential that nutritional interventions have in modulating this balance.
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Influence of vitamin C and vitamin E on redox signaling: Implications for exercise adaptations.
TL;DR: Direct evidence for interference of ascorbate and α-tocopherol with exercise-induced ROS/RNS production is lacking; theoretical analysis reveals that both antioxidants are unlikely to have a major impact on exercise- induced redox signaling; and it is worth considering alternate redox-independent mechanisms.
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Exercise redox biochemistry: Conceptual, methodological and technical recommendations.
TL;DR: Investigators are recommended to consider chemical heterogeneity, use redox-active compounds judiciously, abandon flawed assays, carefully prepare samples and assay buffers, consider repair/metabolism, use multiple biomarkers to assess oxidative damage and redox signalling and provide a unifying set of recommendations.