J
Jamie Peters
Researcher at University of Colorado Denver
Publications - 35
Citations - 2873
Jamie Peters is an academic researcher from University of Colorado Denver. The author has contributed to research in topics: Extinction (psychology) & Nucleus accumbens. The author has an hindex of 16, co-authored 30 publications receiving 2457 citations. Previous affiliations of Jamie Peters include Anschutz Medical Campus & University of Puerto Rico.
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Journal ArticleDOI
Extinction circuits for fear and addiction overlap in prefrontal cortex
TL;DR: Given that the mPFC represents a common node in the extinction circuit for these behaviors, treatments that target this region may help alleviate symptoms of both anxiety and addictive disorders by enhancing extinction memory.
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Infralimbic Prefrontal Cortex is Responsible for Inhibiting Cocaine Seeking in Extinguished Rats
TL;DR: Together, these findings suggest that a neuronal network involving the infralimbic cortex and accumbens shell is recruited by extinction training to suppress cocaine seeking.
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Induction of Fear Extinction with Hippocampal-Infralimbic BDNF
TL;DR: In rats subjected to auditory fear conditioning, BDNF infused into the IL mPFC reduced conditioned fear for up to 48 hours, even in the absence of extinction training, which suggests that BDNF substituted for extinction.
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The group II metabotropic glutamate receptor agonist, LY379268, inhibits both cocaine- and food-seeking behavior in rats.
Jamie Peters,Peter W. Kalivas +1 more
TL;DR: A potential therapeutic role for mGluR2/3 agonists on relapse of cocaine-seeking is supported, however, doses that inhibited cocaine- seeking were only threefold lower than those inhibiting food-seeking, indicating possible unacceptable nonspecific effects.
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Cocaine Increases Actin Cycling: Effects in the Reinstatement Model of Drug Seeking
TL;DR: In this article, the potential involvement of chronic cocaine-induced increases in actin cycling in cocaine addiction was examined using the reinstatement of cocaine seeking in rats previously trained to self-administer cocaine by inhibiting actin polymerization with intra-accumbens latrunculin A or by accelerating actin depolymerization with a LIM-kinase inhibitor.