J
Janusz Moryś
Researcher at Gdańsk Medical University
Publications - 9
Citations - 760
Janusz Moryś is an academic researcher from Gdańsk Medical University. The author has contributed to research in topics: Hypothalamus & Nerve growth factor. The author has an hindex of 5, co-authored 9 publications receiving 462 citations.
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Journal ArticleDOI
BDNF: A Key Factor with Multipotent Impact on Brain Signaling and Synaptic Plasticity.
Przemysław Kowiański,Grazyna Lietzau,Ewelina Czuba,Monika Waśkow,Aleksandra Steliga,Janusz Moryś +5 more
TL;DR: The importance of BDNF for future studies aimed at disclosing mechanisms of activation of signaling pathways, neuro- and gliogenesis, as well as synaptic plasticity is highlighted.
Journal ArticleDOI
Proteasome inhibitors against amelanotic melanoma
TL;DR: It was found that Ab cells died on the mitochondrial pathway of apoptosis and also partially by the caspase-independent way of death, in the first study exploring molecular mechanisms of cell death induced by epoxomicin in melanoma.
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Nicotine-induced CREB and DeltaFosB activity is modified by caffeine in the brain reward system of the rat.
Przemysław Kowiański,Grazyna Lietzau,Grazyna Lietzau,Aleksandra Steliga,Ewelina Czuba,Beata Ludkiewicz,Monika Waśkow,Jan Henryk Spodnik,Janusz Moryś +8 more
TL;DR: Nicotine was the strongest psychostimulant activating ΔFosB-ir in Amg, whereas caffeine - in Hip.
Journal ArticleDOI
Comparison of the influence of two models of mild stress on hippocampal brain-derived neurotrophin factor (BDNF) immunoreactivity in old age rats.
TL;DR: Results indicate that acute FS or HL-OF proved to be a stressor that induces an increase in the density of BDNF-ir pyramidal neurons, which was probably connected with up-regulation of HPA axis activity and short-time memory processing of the stressful situation.
Journal ArticleDOI
The impact of two mild stressors on the nerve growth factor (NGF) immunoreactivity in the amygdala in aged rats compared to adult ones
TL;DR: It is concluded that as far as the influence on NGF‐ir cells in amygdaloid nuclei is concerned, HL‐OF did not constitute an aggravating factor for rats in the ontogenetic periods studied and upregulation of N GF‐ir neurons predominantly in CeA after chronic FS seems to be neuroprotective.