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Jerzy Stanek

Researcher at Cincinnati Children's Hospital Medical Center

Publications -  87
Citations -  1802

Jerzy Stanek is an academic researcher from Cincinnati Children's Hospital Medical Center. The author has contributed to research in topics: Placenta & Pregnancy. The author has an hindex of 22, co-authored 81 publications receiving 1573 citations. Previous affiliations of Jerzy Stanek include University of Cincinnati Academic Health Center & Memorial Sloan Kettering Cancer Center.

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The frequency and severity of placental findings in women with preeclampsia are gestational age dependent

TL;DR: Placental lesions found in women with preeclampsia compared with normotensive control subjects were evaluated to determine whether the presence of these lesions are related to gestational age at delivery and whether the rates of decidual arteriolopathy, central infarction, and hypermaturity of villi were higher the earlier the gestationalAge at delivery.
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Hypoxic patterns of placental injury: a review.

TL;DR: A comprehensive assessment of the criteria for diagnosing acute and chronic histologic features, patterns, and lesions of placental and fetal hypoxia is presented and clinicopathologic associations and limitations of the use thereof are discussed.
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Proliferative activity of human thyroid cells in various age groups and its correlation with the risk of thyroid cancer after radiation exposure.

TL;DR: Findings of a general decrease in proliferative activity of thyroid cells with age may explain, at least in part, the higher risks of radiation-related thyroid cancer in children compared with adults.
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Placental mesenchymal dysplasia associated with fetal aneuploidy.

TL;DR: Three cases of placental mesenchymal dysplasia (PMD) associated with abnormal karyotype are described and cases reported in the literature are reviewed.
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Occult Placenta Accreta: The Missing Link in the Diagnosis of Abnormal Placentation

TL;DR: Although asymptomatic, OPA features the same histopathology as clinical placenta accreta and may share same pathogenesis, which may include decidual deficiency, abnormal trophoblast/ decidua interaction, and/or hypoxia.