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Jin Sub Choi

Researcher at Yonsei University

Publications -  238
Citations -  6541

Jin Sub Choi is an academic researcher from Yonsei University. The author has contributed to research in topics: Hepatectomy & Hepatocellular carcinoma. The author has an hindex of 38, co-authored 215 publications receiving 5662 citations. Previous affiliations of Jin Sub Choi include University Health System.

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Human hepatocellular carcinomas with "Stemness"-related marker expression: keratin 19 expression and a poor prognosis.

TL;DR: K19 was well correlated with clinicopathologic features of tumor aggressiveness, compared to other stemness‐related proteins, and showed significantly increased EMT‐related protein and mRNA expression, suggesting that they may acquire more invasive characteristics.
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The Prognosis and Survival Outcome of Intrahepatic Cholangiocarcinoma Following Surgical Resection: Association of Lymph Node Metastasis and Lymph Node Dissection with Survival

TL;DR: The regional + α lymph node dissection enhanced the survival in the ICC patients with lymph node metastasis, and the exact nodal status could be confirmed by lymph nodes dissection in the pericholedochal lymph nodes.
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Comparative study of resection and radiofrequency ablation in the treatment of solitary colorectal liver metastases.

TL;DR: HR had better outcomes than RFA for recurrence and survival after treatment of solitary colorectal liver metastases, however, in tumors smaller than 3 cm, RFA can be recommended as an alternative treatment to patients who are not candidates for surgery.
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Primary liver carcinoma of intermediate (hepatocyte-cholangiocyte) phenotype.

TL;DR: Intermediate carcinoma may be a distinct type of primary liver carcinoma, morphologically and phenotypically intermediate between HCC and CC, which originates from transformed hepatic progenitor cells.
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DNA methyltransferase expression and DNA methylation in human hepatocellular carcinoma and their clinicopathological correlation

TL;DR: The results suggest that hepatocarcinogenesis involves an increased expression of DNMT1, DNMT3a andDNMT3b mRNA and a progressive increase in the number of methylated genes from normal liver, chronic hepatitis/cirrhosis to HCC and that an increased in the DNMT2a and DNMT4b mRNA levels in HCCs relative to their non-cancerous tissues may be a predictor of poor survival.