J
Joel B. Kirkpatrick
Researcher at University of Texas Southwestern Medical Center
Publications - 15
Citations - 661
Joel B. Kirkpatrick is an academic researcher from University of Texas Southwestern Medical Center. The author has contributed to research in topics: Gliosis & Antigen. The author has an hindex of 12, co-authored 15 publications receiving 654 citations. Previous affiliations of Joel B. Kirkpatrick include University of Texas System & University of Texas Health Science Center at San Antonio.
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Dexamethasone and severe head injury. A prospective double-blind study.
TL;DR: It is suggested that dexamethasone in either high or low dosages has no significant effect on morbidity and mortality following severe head injury and no significant difference when steroid- and placebo-treated patients are compared.
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Late‐onset Hallervorden‐Spatz disease presenting as familial parkinsonism
TL;DR: A 68-year-old man who died after 13 years of progressive dementia, rigidity, bradykinesia, mild tremor, stooped posture, slow and shuffling gait, dystonia, blepharospasm, apraxia of eyelid opening, anarthria, aphonia, and incontinence is studied.
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Multiple System Atrophy: The Putative Causative Role of Environmental Toxins
TL;DR: The case studied pathologically demonstrated extensive advanced glial changes, including glial cytoplasmic inclusions in deep cerebellar white matter, brainstem, cortex (superior frontal, insula) and putamen, with notable cell loss and depigmentation of the substantia nigra and locus ceruleus.
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Joseph disease and Huntington disease: Protein patterns in fibroblasts and brain
TL;DR: Two identical classes of protein changes were present in two nosologically separate autosomal dominant neurological disorders, Joseph disease and HD, and may reflect a biochemical correlation of gliosis and neuronal disease.
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Myoclonus, ataxia, and hypoventilation: Response to L-5-hydroxytryptophan
TL;DR: Both hypoventilation and myoclonus were suppressed for 4 years with L-5-hydroxytryptophan and carbidopa in a patient with a syndrome of progressive myOClonus, ataxia, central neurogenic hypovENTilation, mental retardation, motor neuropathy, and morphologic mitochondrial abnormalities in skeletal muscle.