J
John A. Auchampach
Researcher at Medical College of Wisconsin
Publications - 115
Citations - 6722
John A. Auchampach is an academic researcher from Medical College of Wisconsin. The author has contributed to research in topics: Adenosine receptor & Adenosine. The author has an hindex of 42, co-authored 106 publications receiving 6338 citations. Previous affiliations of John A. Auchampach include University of Virginia & University of Louisville.
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Journal ArticleDOI
Blockade of ATP-sensitive potassium channels prevents myocardial preconditioning in dogs.
TL;DR: The results suggest that myocardial preconditioning in the canine heart is mediated by activation of KATP channels and that these channels may serve an endogenous myocardium protective role.
Journal ArticleDOI
Inhibition of an equilibrative nucleoside transporter by cannabidiol: A mechanism of cannabinoid immunosuppression
TL;DR: It is demonstrated that CBD has the ability to enhance adenosine signaling through inhibition of uptake and provide a non-cannabinoid receptor mechanism by which CBD can decrease inflammation.
Journal ArticleDOI
Adenosine A1 receptors, KATP channels, and ischemic preconditioning in dogs
TL;DR: It is suggested that activation of adenosine A1 receptors produces myocardial preconditioning in the canine heart by opening KATP channels.
Journal ArticleDOI
Blockade of ischaemic preconditioning in dogs by the novel ATP dependent potassium channel antagonist sodium 5-hydroxydecanoate.
TL;DR: The results further strengthen the hypothesis that activation of myocardial KATP channels is involved in the mechanism of ischaemic preconditioning in dogs.
Journal ArticleDOI
PKC-dependent activation of p44/p42 MAPKs during myocardial ischemia-reperfusion in conscious rabbits.
Peipei Ping,Jun Zhang,Xinan Cao,Richard C. Li,Deying Kong,Xian Liang Tang,Yumin Qiu,Srinivas Manchikalapudi,John A. Auchampach,Richard G. Black,Roberto Bolli +10 more
TL;DR: The results demonstrate that ischemic PC induces a rapid activation of p44 and p42 MAPKs in hearts of conscious rabbits and suggest that p44/p42MAPKs contribute to PKC-ε-mediated protection against simulated ischemia.