J
Jonathan D. Cherry
Researcher at University of Rochester
Publications - 21
Citations - 1725
Jonathan D. Cherry is an academic researcher from University of Rochester. The author has contributed to research in topics: Medicine & Chronic traumatic encephalopathy. The author has an hindex of 5, co-authored 6 publications receiving 1286 citations.
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Journal ArticleDOI
Neuroinflammation and M2 microglia: the good, the bad, and the inflamed.
TL;DR: The multiple possible activation states microglia can be polarized to are examined and particular attention is given to utilizing M2 microglial polarization as a potential therapeutic option in treating diseases.
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Galactic Cosmic Radiation Leads to Cognitive Impairment and Increased Aβ Plaque Accumulation in a Mouse Model of Alzheimer’s Disease
Jonathan D. Cherry,Bin Liu,Jeffrey L. Frost,Cynthia A. Lemere,Jacqueline P. Williams,John A. Olschowka,M. Kerry O'Banion +6 more
TL;DR: The results show for the first time that HZE particle radiation can increase Aβ plaque pathology in an APP/PS1 mouse model of Alzheimer’s disease.
Journal ArticleDOI
Arginase 1+ microglia reduce Aβ plaque deposition during IL-1β-dependent neuroinflammation
TL;DR: It is suggested that Arg1+ microglia are involved in Aβ plaque reduction during sustained, IL-1β-dependent neuroinflammation, opening up possible new avenues for immunomodulatory therapy of AD.
Journal ArticleDOI
Are "resting" microglia more "m2"?
TL;DR: It is shown that microglia are not simply macrophages that have migrated into the brain; rather, they are a distinct cell type.
Journal ArticleDOI
Aryl hydrocarbon receptor deletion in cerebellar granule neuron precursors impairs neurogenesis.
Daniel P. Dever,Zachariah O. Adham,Bryan D. Thompson,Matthieu Genestine,Jonathan D. Cherry,John A. Olschowka,Emanuel DiCicco-Bloom,Lisa A. Opanashuk +7 more
TL;DR: The results suggest that AhR activity plays a role in regulating granule neuron number and differentiation, possibly by coordinating this GNP developmental transition, and provide novel insights for understanding the normal roles of AhR signaling during cerebellar granule cell neurogenesis.