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Jonathan W. Said

Researcher at University of California, Los Angeles

Publications -  446
Citations -  27211

Jonathan W. Said is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Lymphoma & Cancer. The author has an hindex of 78, co-authored 437 publications receiving 25399 citations. Previous affiliations of Jonathan W. Said include University of California & Sanford-Burnham Institute for Medical Research.

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Kaposi's Sarcoma–Associated Herpesvirus-Like DNA Sequences in AIDS-Related Body-Cavity–Based Lymphomas

TL;DR: A high degree of conservation of KSHV sequences in Kaposi's sarcoma and in the eight lymphomas suggests the presence of the same agent in both lesions, suggesting that a novel herpesvirus has a pathogenic role in AIDS-related body-cavity-based lymphomas.
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Ligands for peroxisome proliferator-activated receptorgamma and retinoic acid receptor inhibit growth and induce apoptosis of human breast cancer cells in vitro and in BNX mice.

TL;DR: The combination of TGZ and ATRA synergistically and irreversibly inhibits growth and induces apoptosis of MCF7 breast cancer cells and the combination may provide a novel, nontoxic and selective therapy for human breast cancers.
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Inactivation of the tumor suppressor PTEN/MMAC1 in advanced human prostate cancer through loss of expression

TL;DR: It is demonstrated that loss of PTEN/MMAC1 expression occurs frequently in advanced prostate cancer by using a series of recently derived xenografts free of normal human cells and a PTEN/.
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Improved Prognostication of Renal Cell Carcinoma Using an Integrated Staging System

TL;DR: The data suggests that UISS is an important prognostic tool for counseling patients with various stages of kidney cancer and is superior to stage alone in differentiating patients' survival.
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Ligand for Peroxisome Proliferator-activated Receptor γ (Troglitazone) Has Potent Antitumor Effect against Human Prostate Cancer Both in Vitro and in Vivo

TL;DR: Results suggest that troglitazone may be a useful therapeutic agent for the treatment of prostate cancer, especially in the setting of low disease burden.