Jos J. Eggermont

TL;DR: Downregulation of intracortical inhibition induced by damage to the cochlea or to auditory projection pathways highlights neural processes that underlie the sensation of phantom sound.

TL;DR: The results indicate that some areas of the brain activated by sound stimulation have a maturational time course that extends into adolescence and raises the possibility that the emergence of adult-like auditory processing skills may be governed by the same maturing neural processes that affect AEP latency and amplitude.

TL;DR: Evidence is considered that deafferentation of tonotopically organized central auditory structures leads to increased neuron spontaneous firing rates and neural synchrony in the hearing loss region, which covers the frequency spectrum of tinnitus sounds.

TL;DR: Changes in spontaneous activity, recorded over 15-min periods before, immediately after and within hours after an acute acoustic trauma, were studied in primary auditory cortex of ketamine-anesthetized cats, focusing on the spontaneous firing rate (SFR), the peak cross-correlation coefficient (rho) and burst-firing activity.

TL;DR: Evaluated central auditory system maturation based dipole modeling of multi-electrode long-latency AEPs recordings demonstrated the independence of the T-complex components, represented in the radial dipoles, from the P1, N1b, and P2 components, contained in the tangentially oriented dipole sources.