J
José J. Valdés-Aguayo
Researcher at Autonomous University of Zacatecas
Publications - 3
Citations - 33
José J. Valdés-Aguayo is an academic researcher from Autonomous University of Zacatecas. The author has contributed to research in topics: Internal medicine & Mitochondrial DNA. The author has an hindex of 1, co-authored 2 publications receiving 10 citations.
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Journal ArticleDOI
The role of close contacts of COVID-19 patients in the SARS-CoV-2 transmission: an emphasis on the percentage of nonevaluated positivity in Mexico.
Margarita L Martinez-Fierro,Jorge Rios-Jasso,Idalia Garza-Veloz,Lucia Reyes-Veyna,Rosa Maria Cerda-Luna,Iliana Duque-Jara,Maribel Galvan-Jimenez,Leticia A Ramirez-Hernandez,Andres Morales-Esquivel,Yolanda Ortiz-Castro,José R. Gutiérrez-Camacho,José J. Valdés-Aguayo,José R. Vargas-Rodríguez +12 more
TL;DR: The screening for SARS-CoV-2 in close contacts of COVID-19 positive patients should be encouraged to avoid spreading the infection and the expansion of the disease.
Journal ArticleDOI
Mitochondria and Mitochondrial DNA: Key Elements in the Pathogenesis and Exacerbation of the Inflammatory State Caused by COVID-19.
José J. Valdés-Aguayo,Idalia Garza-Veloz,Jose I. Badillo-Almaraz,Sofía Bernal-Silva,Maria C. Martínez-Vázquez,Vladimir Juárez-Alcalá,José R. Vargas-Rodríguez,María L. Gaeta-Velasco,Carolina González-Fuentes,Lorena Avila-Carrasco,Margarita L Martinez-Fierro +10 more
TL;DR: In this article, the importance of mitochondria in inflammatory pathologies, besides providing energy, is associated with the release of mitochondrial damage products such as mt-DNA, which may perpetuate inflammation.
Journal ArticleDOI
Hyperglycemia and Angiotensin-Converting Enzyme 2 in Pulmonary Function in the Context of SARS-CoV-2 Infection
José R. Vargas-Rodríguez,Idalia Garza-Veloz,Virginia Flores-Morales,Jose I. Badillo-Almaraz,María del Refugio Rocha-Pizaña,José J. Valdés-Aguayo,Margarita L Martinez-Fierro +6 more
TL;DR: This document investigated the current evidence related with ACE2 as a key element within the pathophysiological mechanism related with hyperglycemia extrapolating it to context of SARS-CoV-2 infection and its relationship with worse prognosis and death for COVID-19.