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José Luis Sánchez-Quesada

Researcher at Hospital de Sant Pau

Publications -  137
Citations -  3675

José Luis Sánchez-Quesada is an academic researcher from Hospital de Sant Pau. The author has contributed to research in topics: Lipoprotein & Apolipoprotein B. The author has an hindex of 34, co-authored 122 publications receiving 3294 citations. Previous affiliations of José Luis Sánchez-Quesada include Autonomous University of Barcelona & Carlos III Health Institute.

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Response of oxidative stress biomarkers to a 16-week aerobic physical activity program, and to acute physical activity, in healthy young men and women

TL;DR: Regular PA increases endogenous antioxidant activity and LDL resistance to oxidation, and decreases oxidized LDL concentration; 30 min of aerobic PA decreases P-GR and B-GSH-Px activity in the first 30-60 min with a posterior recovery.
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Electronegative low-density lipoprotein.

TL;DR: It is suggested that LDL(-) could contribute to atherogenesis via several mechanisms, including proinflammatory, proapoptotic and antiangiogenesis properties.
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Human Apolipoprotein A-II Enrichment Displaces Paraoxonase From HDL and Impairs Its Antioxidant Properties A New Mechanism Linking HDL Protein Composition and Antiatherogenic Potential

TL;DR: In conclusion, overexpression of human apoA-II in mice impairs the ability of HDL to protect apoB-containing lipoproteins from oxidation, which could explain in part why PON1 is mostly found in HDL particles with apo a-I and without apo A-II, as well as the poor antiatherogenic properties of apo-II–rich HDL.
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Electronegative LDL From Normolipemic Subjects Induces IL-8 and Monocyte Chemotactic Protein Secretion by Human Endothelial Cells

TL;DR: The results indicate that LDL(−) shows proinflammatory activity on ECs and may contribute to early atherosclerotic events.
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Platelet-Activating Factor Acetylhydrolase Is Mainly Associated With Electronegative Low-Density Lipoprotein Subfraction

TL;DR: The high PAF‐AH activity observed in LDL(−) could be related to the proinflammatory activity of these lipoproteins toward cultured endothelial cells.