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Luis M. Vilá

Researcher at University of Puerto Rico, Medical Sciences Campus

Publications -  259
Citations -  14123

Luis M. Vilá is an academic researcher from University of Puerto Rico, Medical Sciences Campus. The author has contributed to research in topics: Lupus erythematosus & Systemic lupus erythematosus. The author has an hindex of 61, co-authored 247 publications receiving 12798 citations. Previous affiliations of Luis M. Vilá include Hofstra University & University of Alabama at Birmingham.

Papers
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Journal ArticleDOI

Human polymorphism at microRNAs and microRNA target sites.

Liuqing Yang, +168 more
- 01 Jan 2013 - 
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A large-scale replication study identifies TNIP1, PRDM1, JAZF1, UHRF1BP1 and IL10 as risk loci for systemic lupus erythematosus

Vesela Gateva, +35 more
- 18 Oct 2009 - 
TL;DR: The results expand the number of confirmed and candidate SLE susceptibility loci and implicate several key immunologic pathways in SLE pathogenesis.
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Effect of hydroxychloroquine on the survival of patients with systemic lupus erythematosus: data from LUMINA, a multiethnic US cohort (LUMINA L)

Graciela S. Alarcón, +7 more
- 01 Sep 2007 - 
TL;DR: Hydxychloroquine, which overall is well tolerated by patients with SLE, has a protective effect on survival which is evident even after taking into consideration the factors associated with treatment decisions.
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Systemic lupus erythematosus in three ethnic groups: XVI. Association of hydroxychloroquine use with reduced risk of damage accrual.

Barri J. Fessler, +8 more
- 01 May 2005 - 
TL;DR: Findings indicate that, after adjustment for propensity to receive HCQ, HCQ usage is independently associated with a reduced risk of damage accrual in SLE patients who had not yet accrued damage at the time of treatment initiation.
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Klinefelter's syndrome (47,XXY) in male systemic lupus erythematosus patients: Support for the notion of a gene-dose effect from the X chromosome

R. Hal Scofield, +16 more
- 01 Aug 2008 - 
TL;DR: The frequency of Klinefelter's syndrome (47,XXY), often subclinical, is increased in men with SLE by approximately 14-fold compared with its prevalence in men without SLE, consistent with the notion that SLE susceptibility is partly explained by an X chromosome gene-dose effect.