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Josipa Kuleš

Researcher at University of Zagreb

Publications -  61
Citations -  711

Josipa Kuleš is an academic researcher from University of Zagreb. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 13, co-authored 46 publications receiving 456 citations.

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Library-based display technologies: where do we stand?

TL;DR: Fundamental technological features are assessed and some recent advances and applications of display technologies are pointed out to address some of the disadvantages of peptides and nucleotides such as their low affinity, low stability, high immunogenicity and difficulty to cross membranes.
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Identification of serum biomarkers in dogs naturally infected with Babesia canis canis using a proteomic approach.

TL;DR: A number of differentially expressed serum proteins involved in inflammation mediated acute phase response, complement and coagulation cascades, apolipoproteins and vitamin D metabolism pathway were identified in dogs with babesiosis.
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Quantitative proteomics using tandem mass tags in relation to the acute phase protein response in chicken challenged with Escherichia coli lipopolysaccharide endotoxin

TL;DR: In this article, the changes in the plasma proteome in chickens challenged with lipopolysaccharide (LPS), a bacterial endotoxin known to stimulate the host innate immune system, were quantified using a quantitative Tandem Mass Tags approach using a Q-Exactive-Plus mass-spectrometer.
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Prevalence and Geographic Distribution of Vector-Borne Pathogens in Apparently Healthy Dogs in Croatia.

TL;DR: The seroprevalence against Babesia canis, Anaplasma phagocytophilum, Borrelia burgdorferi sensu lato, Leishmania infantum, Dirofilaria immitis, and EhrlichiaCanis in dogs in Croatia was investigated in the first comprehensive survey of VBP seropositivity conducted in Croatia.
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Markers of coagulation activation, endothelial stimulation, and inflammation in dogs with babesiosis.

TL;DR: A proinflammatory condition in babesiosis appears to influence endothelial dysfunction and hemostatic activity, and sequestered blood cells can obstruct blood flow in small vessels, promote an inflammatory state, and could increase the severity of babesia infections.