J
Joyce M. Koenig
Researcher at Saint Louis University
Publications - 60
Citations - 1797
Joyce M. Koenig is an academic researcher from Saint Louis University. The author has contributed to research in topics: Neutropenia & Cord blood. The author has an hindex of 22, co-authored 57 publications receiving 1633 citations. Previous affiliations of Joyce M. Koenig include Case Western Reserve University & University of Utah.
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Journal ArticleDOI
Incidence, neutrophil kinetics, and natural history of neonatal neutropenia associated with maternal hypertension.
TL;DR: Kinetic investigations of circulating, marginated, storage, and proliferative neutrophils and their progenitors suggested that the neutropenia was the result of diminished neutrophil production.
Journal Article
Diminished lectin-, epidermal growth factor-, complement binding domain-cell adhesion molecule-1 on neonatal neutrophils underlies their impaired CD18-independent adhesion to endothelial cells in vitro.
Donald C. Anderson,O Abbassi,Takashi Kei Kishimoto,Joyce M. Koenig,Larry V. McIntire,C. W. Smith +5 more
TL;DR: In comparative studies, endothelial cell adhesion of unstimulated cord blood or adult control neutrophils (assessed under conditions of flow) was directly related to levels of neutrophil surface LECAM-1.
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Anthrax Lethal Toxin Paralyzes Neutrophil Actin-Based Motility
Russell L. During,Wei Li,Binghua Hao,Joyce M. Koenig,David S. Stephens,Conrad P. Quinn,Frederick S. Southwick +6 more
TL;DR: It is concluded that 2 h of exposure to LT markedly impairs PMN actin assembly, and reductions in actin filament content are accompanied by a profound paralysis of PMN chemotaxis.
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Group B Streptococcus and Early-Onset Sepsis in the Era of Maternal Prophylaxis
TL;DR: Improved eradication of GBS colonization and disease may involve universal screening in conjunction with rapid diagnostic technologies or other novel approaches, and modern technologies, such as those involving proteomics and genomic sequencing, are likely to hasten the development of a universal vaccine against GBS.
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The mechanism responsible for diminished neutrophil production in neonates delivered of women with pregnancy-induced hypertension.
TL;DR: This common maternal-fetal disorder is associated with an inhibitor of neutrophil production, which is elaborated by the placenta and present in cord blood serum.