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Juan A. Vallejo

Researcher at University of A Coruña

Publications -  39
Citations -  989

Juan A. Vallejo is an academic researcher from University of A Coruña. The author has contributed to research in topics: Acinetobacter baumannii & Virulence. The author has an hindex of 18, co-authored 33 publications receiving 804 citations. Previous affiliations of Juan A. Vallejo include University of Santiago de Compostela.

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Biological Cost of Different Mechanisms of Colistin Resistance and Their Impact on Virulence in Acinetobacter baumannii

TL;DR: While Colistin resistance in A. baumannii confers a clear selective advantage in the presence of colistin treatment, it causes a noticeable cost in terms of overall fitness and virulence, with a more striking reduction associated with LPS loss than with phosphoethanolamine addition.
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Improved Cardiac Function in Patients With Obstructive Jaundice After Internal Biliary Drainage: Hemodynamic and Hormonal Assessment

TL;DR: Increased plasma levels of natriuretic peptides in patients with obstructiveJaundice may reflect a subclinical myocardial dysfunction correlating with the degree of jaundice, and after internal biliary drainage, there is a measurable improvement of cardiac function.
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Short communication: A comparative analysis of recombinant chymosins

TL;DR: It is proposed that recombinant goat chymosin represents a serious alternative to recombinant bovine chymOSin for use in the cheesemaking industry.
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The FhaB/FhaC two-partner secretion system is involved in adhesion of Acinetobacter baumannii AbH12O-A2 strain

TL;DR: Scanning electron microscopy and adhesion experiments demonstrated that inactivation of the AbFhaB/FhaC system significantly decreases bacterial attachment to A549 alveolar epithelial cells, and it has been demonstrated that this 2-partner secretion system is involved in fibronectin-mediated adherence of the A. baumannii AbH12O-A2 isolate.
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Analysis of the role of the LH92_11085 gene of a biofilm hyper-producing Acinetobacter baumannii strain on biofilm formation and attachment to eukaryotic cells.

TL;DR: Scanning electron microscopy and biofilm assays showed that inactivation of LH92_11085 gene significantly reduced bacterial attachment to A549 cells and bio film formation on plastic, respectively, indicating the potential role this LH92-110 85 gene could play in the pathobiology of A baumannii.