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Juan L. Arqués

Researcher at Spanish National Research Council

Publications -  62
Citations -  2562

Juan L. Arqués is an academic researcher from Spanish National Research Council. The author has contributed to research in topics: Nisin & Bifidobacterium. The author has an hindex of 27, co-authored 56 publications receiving 2080 citations. Previous affiliations of Juan L. Arqués include Norwich University & Norwich Research Park.

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Antimicrobial activity of lactic acid bacteria in dairy products and gut: effect on pathogens.

TL;DR: The food industry seeks alternatives to satisfy consumer demands of safe foods with a long shelf-life able to maintain the nutritional and organoleptic quality and the application of antimicrobial compounds-producing protective cultures may provide an additional parameter of processing to improve the safety and ensure food quality, keeping or enhancing its sensorial characteristics.
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Bioactivation of Phytoestrogens: Intestinal Bacteria and Health.

TL;DR: The transformation of isoflavones, ellagitanins, and lignans by intestinal microbiota is essential to be protective against certain chronic diseases, as cancer, cardiovascular disease, osteoporosis, and menopausal symptoms.
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Antimicrobial activity of pediocin-producing Lactococcus lactis on Listeria monocytogenes, Staphylococcus aureus and Escherichia coli O157:H7 in cheese

TL;DR: Because of the antimicrobial activity of pediOCin PA-1, its production in situ by strains of LAB growing efficiently in milk would extend the application of this bacteriocin in cheese manufacture.
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Oral delivery of Lactobacillus casei Shirota modifies allergen-induced immune responses in allergic rhinitis.

TL;DR: The gut microbiota has been implicated in the pathogenesis of allergic disorders, suggesting beneficial interactions between the intestinal immune system and specific bacterial strains, and lactobacilli are currently present in many probiotic supplements.
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Microbial Metabolite Signaling Is Required for Systemic Iron Homeostasis.

TL;DR: Gut microbiota produce metabolites that suppress hypoxia-inducible factor 2α (HIF-2α) a master transcription factor of intestinal iron absorption and increase the iron-storage protein ferritin, resulting in decreased intestine iron absorption by the host.