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Judith S. Gavaler

Researcher at University of Pittsburgh

Publications -  139
Citations -  7643

Judith S. Gavaler is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Liver transplantation & Liver disease. The author has an hindex of 49, co-authored 139 publications receiving 7507 citations. Previous affiliations of Judith S. Gavaler include National Institutes of Health & University of Barcelona.

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Gastrointestinal transit the effect of the menstrual cycle

TL;DR: Gastrointestinal transit time as well as serum estradiol and progesterone levels were measured in 15 normally menstruating women twice during their menstrual cycle, once in the follicular phase and onset of menses at the expected time documenting ovulatory cycles.
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Alcohol-induced testicular atrophy. An experimental model for hypogonadism occurring in chronic alcoholic men.

TL;DR: In this article, an animal model was developed to examine perturbation in gonadal function that occurs in man as a consequence of chronic ethanol ingestion and confirms previous data which suggest that ethanol is a primary testicular toxin.
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Response to interferon α therapy is influenced by the iron content of the liver

TL;DR: No differences between responders and non- responders were evident for the type of hepatitis, age, initial alanine aminotransferase, serum iron, total iron binding capacity, %sat, or ferritin, and the hepatic iron content of non-responders was almost twice that of responders.
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Orthotopic liver transplantation for alcoholic liver disease.

TL;DR: O orthotopic liver transplantation is a therapeutic option that should be considered for individuals with end‐stage alcoholic liver disease who desire such therapy and demonstrate that alcoholic patients can be transplanted successfully and achieve good health not significantly different from that of individuals transplanted for other causes.
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Heartburn of pregnancy.

TL;DR: Data are consistent with earlier studies in women ingesting oral contraceptives and provide support for the thesis that the progressive increase in plasma progesterone alone or in combination with estrogens that occurs during pregnancy is responsible for the reduction of lower esophageal sphincter pressure which allows esophagal reflux to occur with the resultant development of symptomatic heartburn.