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Jukka S. Jurvelin

Researcher at University of Eastern Finland

Publications -  423
Citations -  21884

Jukka S. Jurvelin is an academic researcher from University of Eastern Finland. The author has contributed to research in topics: Cartilage & Bone mineral. The author has an hindex of 77, co-authored 423 publications receiving 20441 citations. Previous affiliations of Jukka S. Jurvelin include University of Bern & Utrecht University.

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Physical activity slows femoral bone loss but promotes wrist fractures in postmenopausal women: a 15-year follow-up of the OSTPRE study

TL;DR: Regular PA of at least 1½ hours per week does not seem to increase the risk of other fractures and might significantly decrease proximal femur bone loss among peri‐ and postmenopausal women.
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Effect of Alendronate on Periprosthetic Bone Loss After Total Knee Arthroplasty: A One-Year, Randomized, Controlled Trial of 19 Patients

TL;DR: The results indicate that oral alendronate reduces early postoperative periprosthetic bone loss significantly, and this therapeutic strategy may improve the results and longevity of primary total knee arthroplasties.
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Factors affecting bone loss around menopause in women without HRT: a prospective study

TL;DR: The beginning of menopause is accompanied by significant bone loss, which decreases in later menopausal women, and few other physiological and life-style factors were found to significantly contribute to this phenomenon.
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Compressive and tensile properties of articular cartilage in axial loading are modulated differently by osmotic environment.

TL;DR: The fibril-reinforced poroelastic model with stiff, nonlinear collagen fibrils predicted the experimentally measured compression-tension nonlinearity of cartilage and proteoglycan (PG) and collagen contents are primary determinants of the compressive and tensile properties of Cartilage.
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Periprosthetic femoral bone loss after total knee arthroplasty: 1-year follow-up study of 69 patients

TL;DR: Bone loss was most rapid during the first 3 months after TKA, and it is suggested that the observed periprosthetic bone loss was mainly the result of prosthesis-related stress-shielding.