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Julieta Aylen Schander

Researcher at University of Buenos Aires

Publications -  12
Citations -  53

Julieta Aylen Schander is an academic researcher from University of Buenos Aires. The author has contributed to research in topics: Offspring & Pregnancy. The author has an hindex of 3, co-authored 10 publications receiving 24 citations.

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Progesterone modulates the LPS-induced nitric oxide production by a progesterone-receptor independent mechanism.

TL;DR: Progesterone downregulated LPS-induced nitric oxide production by a progesterone receptor-independent mechanism, suggesting a possible participation of glucocorticoid receptors in at least some of the anti-inflammatory effects of progester one.
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The enrichment of maternal environment prevents pre-term birth in a mice model.

TL;DR: It is proposed that designing an EE protocol for humans could be a noninvasive and preventive therapy for pregnancy complications, averting pre-term birth occurrence and dreaded sequelae that are present in the offspring born to soon.
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Maternal obesogenic diet combined with postnatal exposure to high-fat diet induces metabolic alterations in offspring.

TL;DR: Evidence is presented that the offspring from mothers exposed to a HFD showed increased acetylation levels of histone 3 on lysine 9 (H3K9) in the liver at postnatal Day 1, whereas the levels of acetylated levels of H4K16, dimethylation of H3K27, and trimethylated levels showed no change.
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PGE2 displays immunosuppressive effects during human active tuberculosis.

TL;DR: It is demonstrated that PGE2 exerts a potent immunosuppressive action during the immune response of the human host against Mycobacterium tuberculosis (Mtb) infection and the potential of this lipid mediator as a tool to improve anti-TB treatment is highlighted.
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A role for the endocannabinoid system in premature luteal regression and progesterone withdrawal in lipopolysaccharide-induced early pregnancy loss model.

TL;DR: The results suggest a role for the eCS mediating LPS-induced deleterious effects on reproductive tissues as well as new insights in the role of the endocannabinoid system in the physiopathology of reproduction.