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Jun-li Liu

Researcher at Xinxiang Medical University

Publications -  5
Citations -  16

Jun-li Liu is an academic researcher from Xinxiang Medical University. The author has contributed to research in topics: Biology & Chemistry. The author has an hindex of 1, co-authored 1 publications receiving 6 citations.

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Journal ArticleDOI

Indirubin, a small molecular deriving from connectivity map (CMAP) screening, ameliorates obesity-induced metabolic dysfunction by enhancing brown adipose thermogenesis and white adipose browning

TL;DR: Results clearly show that as an effective BAT (as well as beige cells) activator, indirubin may have a protective effect on the prevention and treatment of obesity and its complications.
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Proteomics-based evaluation of the mechanism underlying vascular injury via DNA interstrand crosslinks, glutathione perturbation, mitogen-activated protein kinase, and Wnt and ErbB signaling pathways induced by crotonaldehyde

TL;DR: In this paper , the authors used proteomics to systematically characterize the presently unclear molecular mechanism of vascular injury and to identify new related targets or signaling pathways after exposure to Crotonaldehyde.
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Proteomics-based evaluation of the mechanism underlying vascular injury via DNA interstrand crosslinks, glutathione perturbation, mitogen-activated protein kinase, and Wnt and ErbB signaling pathways induced by crotonaldehyde

TL;DR: In this paper , the authors used proteomics to systematically characterize the presently unclear molecular mechanism of vascular injury and to identify new related targets or signaling pathways after exposure to Crotonaldehyde.
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Metabolic crosstalk between thermogenic adipocyte and cancer cell: Dysfunction and therapeutics.

TL;DR: The role of thermogenic adipose tissue in malignant cancer behavior has been largely overlooked as mentioned in this paper , however, emerging evidence suggests that beige/brown adipocytes play a key role in the development and progression of various cancers.
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Thioredoxin 1 overexpression attenuated diabetes‐induced endoplasmic reticulum stress in Müller cells via apoptosis signal‐regulating kinase 1

TL;DR: Wang et al. as discussed by the authors used DM mouse and high glucose (HG)cultured human Müller cells as models to clarify the effect of Trx1 on ERS and the underlying mechanism.