Kaoru Asada

TL;DR: Elevated LV end-diastolic pressure is associated with a decrease of peak LA wall strain in the longitudinal direction during LV systole, and this correlation was also significant in patients with preservedLV systolic function.

TL;DR: Elevated BNP levels are independently associated with higher RDW in patients with CAD and neurohumoral activation may be a mechanistic link between increased RDW and adverse clinical outcomes in this population.

TL;DR: Increased AI is independently associated with LV diastolic function in patients with known or suspected CAD, and late-systolic load may be a therapeutic target to improve LV diastsolic abnormalities in this population.

TL;DR: In patients with PSF, LV apical wall motion abnormality was less frequently observed in those with inertia force than in those without, suggesting an absence of inertia force is one of the causes of isolated diastolic dysfunction in patients with CAD.

TL;DR: Using plasma BNP level and with the combination of B NP level and mitral annular velocity during early diastole, invasively proved isolated LV diastolic dysfunction without heart failure could be identified in patients with coronary artery disease.