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Kara L. Kerr

Researcher at University of Tulsa

Publications -  32
Citations -  1008

Kara L. Kerr is an academic researcher from University of Tulsa. The author has contributed to research in topics: Nociceptive flexion reflex & Nociception. The author has an hindex of 17, co-authored 29 publications receiving 796 citations. Previous affiliations of Kara L. Kerr include McGovern Institute for Brain Research.

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Depression-Related Increases and Decreases in Appetite: Dissociable Patterns of Aberrant Activity in Reward and Interoceptive Neurocircuitry

TL;DR: Depression-related increases in appetite are associated with hyperactivation of putative mesocorticolimbic reward circuitry, while depression-related appetite loss is associated with hypoactivation of insular regions that support monitoring the body's physiological state.
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Altered Insula Activity during Visceral Interoception in Weight-Restored Patients with Anorexia Nervosa

TL;DR: Individuals with AN displayed increased activation during anxious rumination in the dorsal mid-insula, and activation in this region during stomach interoception was correlated with measures of anxiety and psychopathology, which suggests an important mechanism for the disorder.
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Appetite changes reveal depression subgroups with distinct endocrine, metabolic, and immune states

TL;DR: Novel evidence is provided linking aberrations in homeostatic signaling pathways within depression subtypes to the activity of neural systems that respond to food cues and select when, what, and how much to eat is provided.
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A common gustatory and interoceptive representation in the human mid-insula.

TL;DR: healthy volunteers were asked to undergo fMRI while performing tasks involving interoceptive attention to visceral sensations as well as a gustatory mapping task, and analysis of the unsmoothed, high‐resolution fMRI data confirmed shared representations of gustatory and visceral interoception within the dorsal mid‐insula.
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Pain catastrophizing is related to temporal summation of pain but not temporal summation of the nociceptive flexion reflex

TL;DR: Results confirm prior studies that indicate that catastrophizing enhances pain via supraspinal processes rather than spinal processes, and caution is warranted when using pain ratings to infer temporal summation of spinal nociceptive processes.