K
Karel Pardaens
Researcher at Katholieke Universiteit Leuven
Publications - 18
Citations - 741
Karel Pardaens is an academic researcher from Katholieke Universiteit Leuven. The author has contributed to research in topics: Heart failure & Blood pressure. The author has an hindex of 14, co-authored 18 publications receiving 702 citations.
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Influence of demographic, anthropometric and lifestyle characteristics on heart rate and its variability in the population.
TL;DR: Age, gender and/or some lifestyle factors significantly affect heart rate and various components of its variability in the supine position and in response to standing, whereas independent effects of habitual physical activity, smoking habits and body mass index explained no more than 4% of the variance of some measures of heart rate variability.
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Atrial fibrillation is associated with a lower exercise capacity in male chronic heart failure patients
TL;DR: Atrial fibrillation is associated with a 20% lower peak V˙O2 in patients with chronic heart failure, suggesting that preserved atrial contraction or a regular rhythm, or both, are critical to maintain cardiac output and exercise performance.
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Prognostic Value of Invasive Hemodynamic Measurements at Rest and During Exercise in Hypertensive Men
TL;DR: It is concluded that the prognostic importance of blood pressure is related to systemic vascular resistance, which provides prognostic information beyond that available from measurements at rest, particularly for the incidence of cardiovascular events.
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Peak oxygen uptake better predicts outcome than submaximal respiratory data in heart transplant candidates.
TL;DR: Respiratory data during submaximal exercise are significant predictors of outcome in patients with chronic heart failure, but their prognostic power is inferior to that of peak VO(2).
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The pulse pressure-to-stroke index ratio predicts cardiovascular events and death in uncomplicated hypertension
TL;DR: The PP-to-SVi ratio was a significant predictor of fatal and nonfatal cardiovascular events and of all-cause mortality after control for age and gender and its predictive power persisted after additional adjustment for mean arterial pressure and heart rate.