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Kazunori Yamada

Researcher at Kanazawa University

Publications -  83
Citations -  2571

Kazunori Yamada is an academic researcher from Kanazawa University. The author has contributed to research in topics: Kidney disease & IgG4-related disease. The author has an hindex of 21, co-authored 77 publications receiving 2277 citations. Previous affiliations of Kazunori Yamada include University of Geneva.

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Lack of Galactosylation Enhances the Pathogenic Activity of IgG1 but Not IgG2a Anti-Erythrocyte Autoantibodies

TL;DR: Analysis of in vivo complement activation and of the pathogenic activity in mice deficient in C3 or IgG FcRs excluded the implication of mannose-binding lectin-mediated complement activation in the enhanced pathogenic effect of agalactosylated IgG1 anti-erythrocyte autoantibodies.
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IgG4-related skin lesions in a patient with IgG4-related chronic sclerosing dacryoadenitis and sialoadenitis

TL;DR: Although the swollen Lacrimal and parotid gland and itchy subcutaneous erythematous nodules improved rapidly with oral prednisolone at a dose of 20 mg per day, the skin, lacrimal, and parotsid lesions deteriorated simultaneously during steroid tapering and improved after increasing the dosage.
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Pericardial Involvement in IgG4-related Disease

TL;DR: This case indicates that recognizing pericardial complications in autoimmune pancreatitis is important and that CT imaging may be useful for obtaining the diagnosis and providing follow-up of pericARDial lesions in cases of IgG4-RD.
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IgG4-related tubulointerstitial nephritis and hepatic inflammatory pseudotumor without hypocomplementemia.

TL;DR: A 58-year-old Japanese man with epigastralgia underwent contrast-enhanced computed tomography (CT), which revealed multiple low-density lesions in both kidneys and a low density hepatic mass, and Laboratory tests showed an extremely high level of serum IgG4.
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Concurrent presentation of cryptococcal meningoencephalitis and systemic lupus erythematosus.

TL;DR: A patient diagnosed simultaneously with cryptococcal meningoencephalitis and SLE suggests that intrinsic immunological abnormality related to SLE predisposed to opportunistic infections.