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Kenneth M. Rosen

Researcher at University of Illinois at Chicago

Publications -  299
Citations -  13275

Kenneth M. Rosen is an academic researcher from University of Illinois at Chicago. The author has contributed to research in topics: Electrical conduction system of the heart & Tachycardia. The author has an hindex of 65, co-authored 299 publications receiving 13084 citations. Previous affiliations of Kenneth M. Rosen include Mercy Medical Center (Baltimore, Maryland) & Baylor College of Medicine.

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Journal ArticleDOI

Demonstration of Dual A-V Nodal Pathways in Patients with Paroxysmal Supraventricular Tachycardia

TL;DR: These findings provide a basis for reentrance in some patients with reentrant PSVT, as manifest by dual A-V nodal conduction times and refractory periods, and Antegrade failure of the fast pathway with subsequent availability for retrograde conduction could allow A- V nodal reentry.
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The Effects of Cycle Length on Cardiac Refractory Periods in Man

TL;DR: The responses of the human heart to changes in cycle length are generally similar to those previously described in the animal laboratory, which contributes to the understanding of electrocardiographic phenomena such as aberrant conduction.
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Clinical, electrocardiographic and electrophysiologic observations in patients with paroxysmal supraventricular tachycardia.

TL;DR: A mechanism of paroxysmal supraventricular tachycardia could be defined in most patients, and Observations of clinical and electrocardiographic features in these patients should allow prediction of the mechanism of the tachycardsia.
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Cardiac conduction in patients with symptomatic sinus node disease.

TL;DR: In conclusion, abnormalities of conduction and automaticity in specialized tissue in addition to the sinus node are common in patients with symptomatic sinus nodes disease and should be recognized when present, so that the ideal site for permanent pacing may be chosen.
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Left atrial transport function in myocardial infarction. Importance of its booster pump function.

TL;DR: In patients with myocardial infarction, atrial contraction made a large contribution to left ventricular filling and stroke volume irrespective of the type ofleft ventricular functional derangement that was present.